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It is likely that the most well-known function of NADPH oxidase-derived superoxide is the inactivation of NO in the reaction that forms peroxynitrite, which leads to impaired endothelium dependent vasodilation and the uncoupling of endothelial NO synthase (eNOS) to produce additional superoxide

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In conclusion, the relationship between HO-1 and programmed cell death in diabetes deserves further investigation (Fig

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Cao L, Waldon D, Teffera Y, Roberts J et al (2013) Ratios of biliary glutathione disulfide (GSSG) to glutathione (GSH): a potential index to screen drug-induced hepatic oxidative stress in rats and mice