In this study, we provide evidence for regulated ATP release from enteroendocrine L-cells and demonstrate functional purinergic signalling from L-cells to enterocyte and neuronal targets within the colonic mucosa
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We also found that the R152H variant of GPX4 is less susceptible to degradation, revealing the degradation mechanism of the GPX4 protein
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The two products follow different regulatory frameworks, different stability testing protocols, and different shelf-life designations
