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The strongly diabetogenic effect of GCs is caused by increased insulin resistance, driven primarily by increased hepatic gluconeogenesis, lipolysis, and elevated circulating free fatty acids (35)

Without a provider review, these contraindications may not be identified before treatment begins

36 Moreover, GLP-1 RAs improve lipid profiles by decreasing serum cholesterol and triglyceride levels and increasing high-density lipoprotein (HDL) levels contributing to cardiovascular protection

Oral semaglutide and cardiovascular outcomes in patients with type 2 diabetes

Mechanisms of activation of mouse and human enteroendocrine cells by nutrients

Activation of GLP-1R by GLP-1 or other exogenous agonists, including exendin-4 and liraglutide, decreases inflammatory responses in several animal models like rat heart and whole animal model