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BPC-157 operates locally as an **ultra-stable cytoprotectant**: it survives gastric milieu, reactivates the VEGF-receptor-2 pathway at the lesion site, modulates the nitric oxide system (NO / eNOS), upregulates growth hormone receptors on tendon fibroblasts (Chang et al

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These findings suggest that EA treatment mitigates the worsening of symptoms following TBI, potentially by inhibiting TGIF-mediated apoptosis in secondary brain injury post-TBI

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Have severe or unstable medical conditions (e.g., uncontrolled diabetes, end-stage renal disease, significant heart or liver disease) without first consulting a physician