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Nevertheless, degenerative changes were reduced in the AHQG group

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Hyperlysinemia type I is an autosomal recessive disorder that manifests in infancy with nonspecific seizures, hypotonia, or mildly delayed psychomotor development

This correlation with the quantified degeneration area further supported the idea that, in the absence of hepsin, hepatic impairment is more severe during the early stages of APAP exposure

This effect likely relates to the peptide's GABAergic and anti-NMDA activities