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found that Exos released from pericardial adipose tissue interacted with iron regulatory protein 2, helping to protect cardiomyocytes from ferroptosis while maintaining iron homeostasis disrupted by Adipsin [153]

Using a baseline homocysteine level of 9.0 umol/L the researchers found that for every 5.0 umol/L increment increase in homocysteine levels, all-cause mortality increased by 49%, cardiovascular mortality by 50%, cancer mortality by 26%, and deaths from other causes (respiratory, gastrointestinal and central nervous system diseases) by 104%

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This pattern was also observed in people with type 2 diabetes (Figure 5D)

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