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Conversely, reactive astrocytes produce TNF- and IL-6, further enhancing microglial activation and inflammation [141,142,143,144]

In addition, MOTS-c treatment significantly increased the phosphorylation level of AMPK and inhibited the activation of extracellular signal-regulated kinase (ERK), c-Jun N-terminal kinase (JNK), P38, and c-Fos expression induced by formalin (8)

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