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GLP-1 may induce sodium excretion by blocking sodium reabsorption in the proximal tubule through inhibition of sodium-hydrogen exchanger isoform 3 (NHE3) activity [23], as no GLP-1 receptors appear to be present in human kidneys outside of the renal vasculature [19]

1 Depending on the specific molecules half-life, GLP-1 medications can also have a short-release or extended-release action, determining how long their effects last in the body

Once it spots them, glutathione neutralises them before they cause harm

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