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As expected, BSA-PC reduced the phosphorylation of GSK-3 at S9 in SH-SY5Y cells compared to those treated with BSA alone, without altering native GSK-3 protein levels (Fig 5A and 5B)

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These agents primarily exert their effects through specific binding to GLP-1 receptors, thereby stimulating G protein Gs-mediated signaling pathways 2