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The hallmark pathological features of AD include the accumulation of -amyloid (A) peptides forming senile plaques and hyperphosphorylated tau aggregating into neurofibrillary tangles (NFTs), accompanied by synaptic dysfunction, neuronal loss, and chronic neuroinflammation (Agostinho et al., 2010

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10.1182/blood-2017-09-806737 8 AngeliJ

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decreased TUNEL-positive nuclei), and stimulation of myocardial cell proliferation (elevated expression of PCNA and cyclin D1) (Xu et al., 2021)