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As mentioned above, despite the theoretical possibility that GH-RH agonists could stimulate prostate cell proliferation through GH/IGF-1 signaling, in vivo studies have paradoxically shown inhibitory effects on tumor growth due to receptor downregulation

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11,12 On the other hand, AMPs can be internalized into cancer cells via endocytosis mechanisms, leading to apoptosis and the inhibition of autophagy