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Mitophagy, which is the selective degradation of damaged mitochondria, serves a notable role in maintaining mitochondrial homeostasis and regulating ferroptosis

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and, in the context of obesity treatment, differences in dose, titration speed, co-occurring nausea, and baseline oral health may further amplify inter-individual phenotypic variation

A meta-analysis of randomized clinical trials has similarly highlighted significant effects on SBP, with mechanistic links to NO-mediated endothelial repair

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