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Why does the A peptide of Alzheimer share structural similarity with antimicrobial peptides
Elgazzar UB, Ismael I, Ghanema A, Kalaba ZM
This duality poses a critical challenge: therapeutic interventions must be timed to target pathological ferroptosis without abrogating its protective roles in early injury clearance
helps fight cell-damaging free radicals in the body Glutathione regeneration
When telomeres become critically short: Cells stop dividing Tissue regeneration declines Senescent cells accumulate Aging accelerates Researchers consider telomere shortening one of the fundamental hallmarks of aging
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