The status epilepticus induces neuronal LONP1 upregulation, which maintains neuronal survival during prolonged seizures by clearing excessive mitochondrial active caspase-3 and high-mobility group box 1 (HMGB1) [90]
And it turns out that the VDREs in tryptophan hydroxylase 2
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Virus-induced hyper-inflammation with complement activation in kidney tissue could inflict focal segmental glomerulo-sclerosis with glomerular involution and lead to AKI 138
