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CFTR-dependent functional enterocyte maturation in human premature intestinal epithelial models via EV39 or TGF1 and FGF2 To corroborate the EV39- or TGF1 and FGF2-induced enrichment of adult-type enterocyte signatures, we investigated whether these treatments improve epithelial barrier integrity and fatty acid uptake in human premature intestinal epithelial models, with a focus on CFTR dependency

Elevated actin filament-related proteins (e.g., ACTC1, LMOD3) reflect the need for efficient myofibril organisation in FG, but rapid hypertrophy may overwhelm cellular homeostasis, potentially contributing to muscle myopathies like WS 7

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10.1097/CHI.0b013e3181799ee8 J Am Acad Child Adolesc Psychiatry

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