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The findings revealed that irregular activation of HGF/c-MET enhanced TGF- release from cancer cells, reduced CTL penetration at the tumor site, and eventually caused immune evasion

Because cysteine availability is a limiting factor for glutathione synthesis, treatment with N-acetylcysteine may increase glutathione levels and thereby counter oxidative stress, promote redox -regulated cell signaling, and improve immune responses

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PMID: 15261841

Fewer sebaceous glands, lower baseline collagen turnover