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Compounds present in the blood typically undergo an extensive phase II metabolism in the enterocytes and hepatocytes yielding sulfate and glucuronic acid conjugates

To examine the extent to which the phenotypes observed were specifically caused by lower GSH levels, we rescued GSH depletion in liver-specific Gclc KO mice with GSH mono ethyl ester (GSH-ee)

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This may imply conformational changes which don't allow the fragment to accept the helical regularity since disulfide bridges are stronger than the forces stabilizing alpha helix

Enhanced Ingredients