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Vaidyanathan K, Niranjan T, Selvan N, Teo CF, May M, Patel S, et al

In contrast, in non-glucose starved cells, with the presence of 10 mM glucose in all perfusion buffers, no NADP oxidation was observed, even with continuous perfusion of buffer containing 20 M H 2 O 2

The molecular pathways by which mitochondrial derangements act as a unifying pathophysiological link between these conditions are explored in detail, with a particular focus on the oxidative stressinflammation cascade, damage to the vascularneural unit, and systemic metabolic reprogramming

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