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Xinhua Qu, [email protected] Specialty section: This article was submitted to Bone Research, a section of the journal Frontiers in Endocrinology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

In summary, our findings identify MAPKAPK2 as a potentially epigenetically silenced, stress-responsive kinase that mitigates oxidative damage and supports antioxidant defense in vitiligo melanocytes

Its interaction with MI is most developed: MI improves insulin signaling, while melatonin supports granulosa-cell mitochondria, together improving oocyte competence in early studies