Neurodevelopmental disorder-associated SNIP1 mutation E366G reduces binding to P-SF3B1 A homozygous SNIP1 mutation (E366G) found in ~ 35 individuals from the Amish population has been identified as the genetic cause of a neurodevelopmental disorder characterized by hypotonia, craniofacial abnormalities, and seizures (NEDHCS) 47,48
reported that stem cells derived from human exfoliated deciduous teeth (SHEDs) exhibit significant preventive and therapeutic effects on HACE by inhibiting M1-type polarization and promoting M2-type polarization of microglia via the HIF/ERK pathway (59)
Iron deficiency transiently suppresses biliary neuronal nitric oxide synthase
BPC-157 and Digestive Balance in Research Models BPC-157 has been evaluated in extensive preclinical studies involving rodent gut injury models such as NSAID-induced damage, ulcers, and inflammation
As previously mentioned, the associations of vitamin B12 concentrations with NAFLD may differ in populations of different ancestries
Journal of Biological Chemistry, 280 , 3950539509
