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The newly recruited monocytes express a pro-inflammatory phenotype (Ly6C hi ) [103] and there is evidence that during the very early phase of their recruitment (812 h after APAP) these cells can cause a temporary mild aggravation of the injury [108]

Isotretinoin reduces sebum production by 70 to 90% through retinoic acid receptor signaling [1]

Acute respiratory distress syndrome (ARDS) involves neutrophil accumulation in alveolar spaces and NSP release, causing alveolar-capillary barrier damage and exacerbated inflammatory responses (Donnelly et al., 1995)

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