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L-ornithine is also converted into L-arginine, which can enhance the release of growth hormone

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DHODH/CoQH2 axis is an ferroptosis inhibitor independent of the classical GPX4 signaling pathway and based on mitochondrial lipid peroxidation of ferroptosis

Acting as a crucial regulator of the NLRP3 inflammasomecaspase1coagulation axis, VX765 predominantly exerts its therapeutic efficacy via caspase1 inhibition, thus preventing: (1) the assembly of the inflammasome complex, (2) the maturation and secretion of proinflammatory cytokines (especially IL1 and IL6), and (3) GSDMDmediated pyroptotic cell death