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Thus, the acetyl moiety from ALCAR can be oxidized for energy, serve as a precursor for acetylcholine, and be incorporated into amino acid neurotransmitters and lipids in brain [57, 102]

(63) demonstrated that the DIO2 Thr92Ala polymorphism substantially reduces type 2 deiodinase activity, impairs local T4-to-T3 conversion, and is associated with lower serum T3 levels in patients treated with levothyroxine

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In the context of cancer therapy, our results suggest that stimulation of nucleotide synthesis in combination with GSH depletion may be especially toxic

Consequently, direct translation of microbiome-related findings from rodents to humans should be approached with caution