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[67] found that in the HBV transgenic mouse model, cytotoxic T lymphocytes (CTLs) specific for HBcAg are crucial for controlling HBV replication and preventing liver damage in patients with chronic hepatitis B

We also compared the metabolic and phenotypic consequences of genetically impairing 2-OG or glutarate metabolism, and depleted HeLa cells of ABHD11 or GCDH (Fig