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These neural shifts are correlated with subjective reports of reduced craving intensity and improved emotional regulation

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However, when these pathways become sluggish or compromised due to chronic illness, toxins can accumulate and recirculate throughout the bloodstream

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The SLC7A11/GSH/GPx4 axis plays a central role in the cells defense against ferroptosis, with GPx4 utilizing glutathione (GSH) as a cofactor to reduce toxic lipid peroxides at the plasma membrane, thereby protecting the cell from ferroptosis

The second most elevated process was metabolic pathways that encompassed the overrepresentation of nucleotide metabolism, glycolysis, and glutathione metabolism (with almost all primary enzymes of the latter process, e.g., GPX1-3, GSTA1-4 upregulated) (Table S2)