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For example, a double-alanine mutagenesis scan of ECL1 of rGLP-1R identified a residue pair Met 204 Tyr 205 as interacting with GLP-1's N-terminal 8 residues (Lpez de Maturana et al ., 2004)

Furthermore, the activation of GLP-1R signaling in the ventromedial hypothalamus (VMH) can also control energy expenditure by promoting food intake-independent weight loss by inducing brown adipose tissue (BAT) thermogenesis and browning through a sympathetic drive to BAT [6]

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