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however, their overproduction under oxidative stress disturbs the continuity of biological membranes, as well as the basic metabolic processes of cells [110]

For instance, in hepatocellular carcinoma HepG2 cells, CGA was able to prevent the degradation of the extracellular matrix by reducing the expression of MMP-2 and MMP-9, thus inhibiting the migration and invasion of tumor cells [82]

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Of great importance, opposite regulation of key pro-fibrotic marker genes -smooth muscle actin ( ACTA2 ) and connective tissue growth factor ( CTGF ) by aloxistatin in TGF1-stimulated HCFs derived from both ICM and DCM patients could be validated using qPCR (Fig

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