The metabolic kynurenic pathways is likely also implicated in ME/CFS, owing to common mutations in IDO2 (76) such as R248W and Y359STOP (77)
Distinct cysteine residues in Keap1 are required for Keap1-dependent ubiquitination of Nrf2 and for stabilization of Nrf2 by Chemopreventive agents and oxidative stress
Expected timeline: Week 1 to 3: Reduction in inflammation, improved mobility, some pain relief Week 3 to 6: Significant improvement in nerve symptoms, increased functional capacity Week 6 to 12: Continued tissue remodeling, disc structural improvements Month 3 to 6: Full benefits of the protocol become apparent as remodeled tissue matures Understanding proper peptide storage is critical during multi-week protocols
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