By revealing how EMFs spatiotemporally coordinate these axes [Figure 1], we redefine a new therapeutic paradigm: exploiting electromagnetic bioeffects to simultaneously disrupt oncogenic circuits (e.g., Ca 2+ -driven stemness, EMF-mediated mechanisms in cancer pathobiology Additionally, sex hormones (particularly androgens) drive PCa progression by activating oncogenic signaling pathways like AR-dependent transcription
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Thus, postprandial PYY release under normal physiological conditions is likely to be mediated through paracrine and neural mechanisms (123)
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