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doi: 10.3390/ijms22020860 148 ZhangMWuYGaoRChenXChenRChenZ

Glover GH

El control metablico de la glucosa es ms complejo que la exclusiva interaccin entre la insulina y el glucagn

GLUT1, glucose transporter protein 1

In this context, even moderate increases in oxidative demand can favor conditions that increase mitochondrial electron leakage and ROS formation, particularly when respiratory reserve and redox-buffering capacity are reduced