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After reaching 8090% confluency in 23 days, cells were seeded into 12-well plates for in vitro experiments

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Moon JS, Hong JH, Jung YJ, Ferrannini E, Nauck MA, Lim S

Conclusion: PQQ can regulate the pathogenesis of myocardial hypertrophy through the induction of YAP-related anti-ferroptotic activity, highlighting the potential value of PQQ as a novel therapeutic agent capable of slowing or preventing the progression of myocardial hypertrophy and thus delaying the onset of heart failure

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