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However, we have found that removal of mast cells from mixed rat and mouse peritoneal cell populations dramatically depletes NO production, and that residual NO production can be fully accounted for by the low numbers (12%) of contaminating nonmast cells [45,47]

Oral N-acetylcysteine rescues lethality of hepatocyte-specific Gclc-knockout mice, providing a model for hepatic cirrhosis

The direct route means effects are often felt much faster than with oral intake

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