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This may partially explain why no significant associations have been found between homozygosity for the MTHFR thermolabile defect or heterozygosity for mutations in the CBS gene and cardiovascular risk (Folsom et al, 1998

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In the current study, we query the cellular composition and functionality/signaling of hepatoblastoma tumor-adjacent BWS liver compared to hepatoblastoma tumor-adjacent nonBWS liver, as a first step in understanding the mechanisms of BWS overgrowth and liver cancer induction

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