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Adding exogenous glutathione to a drug that already modulates oxidative stress raises a conceptual question: could the combined antioxidant load suppress reactive oxygen species (ROS) to a degree that interferes with beneficial ROS signaling

If it does not, that is a flag to look for other causes (kidney function, MTHFR genetic variants, certain medications)

(eds) Protein Misfolding and Cellular Stress in Disease and Aging

Some are, including GLP-1 receptor agonists (semaglutide, tirzepatide), insulin, and a range of hormone-based therapeutics