current strategies often disrupt normal mitochondrial function in healthy tissues, and biomarkers to stratify patients likely to respond to such therapies (based on mitochondrial ferroptosis signatures) are still absent
In addition, it was shown that the infusion of N-acetylcysteine led to a rapid rise in GSH levels in the brain [46]
doi: 10.1073/pnas.0712185105 170 TohmeSYazdaniHOAl-KhafajiABChidiAPLoughranPMowenKet al
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SOD, GSH and catalase are dominating intracellular antioxidants present or released into the circulation following disease related erythrocyte catastrophe21, their plasma/serum activities are changed by several orders of magnitude than corresponding erythrocyte levels22
