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This is because, originally, scientists investigated BPC-157 in relation to gastric biology and the broader gastrointestinal tract

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ROS has also been implicated as an apoptotic stimulus that triggers the mitochondria to release some signaling molecules crucial for the activation of programmed cell death (97)

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Newer strategies to address these drawbacks like combination therapy (use of structurally different chelating agents) or co-administration of antioxidants have been reported recently