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mTORC1: Mechanistic target of rapamycin complex 1

Additionally, serine-derived metabolites such as glutathione, glycine, cysteine, and SAM support phase II metabolism because they are utilized for conjugation with toxins for toxin metabolism and excretion

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In fact, mutational disruption of the guiding bar did leave to a clear increase in hTrx1- and DTNB-reduction activity of hTrxR1 (Table 2)

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