*Correspondence: Ricardo Tavares Pinheiro, [email protected] This article was submitted to Mood Disorders, a section of the journal Frontiers in Psychiatry Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
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A: Molecularly identical
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Understanding the links between inflammatory mechanisms, tendon homeostasis and resolution of tendon damage will be crucial in developing novel therapeutics for human tendon disease
Furthermore, we verified that celastrol suppresses ferroptosis through the SIRT1/NRF2/GPX4 signalling pathway, suggesting its potential as a renal protective agent and providing novel therapeutic insights for DKD treatment