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Collectively, these findings position GLP-1RAs as modulators of atherosclerotic disease beyond glycemic control, with integrated effects spanning systemic immunometabolism and plaque biology

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however, these therapeutic options are often inefficient, have significant side effects and its prescription depends on the presence of comorbidities (Obrosova et al., 2008

We can actually support and nudge increase of GLP-1 (Glucagon-like peptide-1) naturally, which is the very hormone these medications mimic, through targeted, evidence-based nutritional strategies

In 2012, Dixon et al

Signal transduction of the GLP-1-receptor cloned from a human insulinoma