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Additionally, insulin resistance increases the number of neuritic plaques by affecting tau phosphorylation, widely considered a pivotal pathological hallmark in Alzheimers disease (Wegmann et al., 2021)

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These non-fused peptide data are also inconsistent with the model of receptor activation by the insertion of the peptide N-terminus into the GLP-1R TMD, but they are consistent with the data obtained with the peptide-fused receptors

These systems shorten analytical cycles and flag anomalies during live experiments