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Interestingly, a few years ago it has been published that in flies missense mutations of the SLC25A39 homologue shawn results in accumulation of ROS as well as in mitochondrial dysfunction, synaptic defects, and neurodegeneration

The biphasic viral increase seen in mice can be explained by a partial block of virion production possibly due an early stage of innate immune response, reminiscent of the observed biphasic increase in chimpanzees that was suggested due to an endogenous type I interferon response (ibid)

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Ceriello A, Giugliano D, Quatraro A, Lefebvre PJ