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A reduction in IL-2 cytokine expression can affect not only the proliferation of pro-inflammatory Th1/Th17 cell subsets promoting ALS progression, but can also increase Treg/Th2 subsets that provide anti-inflammatory stimuli resulting in protection during an inflammatory event

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Removing it reduces IGFBP affinity by approximately 90%, increasing the free fraction of the peptide available for IGF1R engagement