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In this article we provide an overview of the pathophysiology and management of NASH, with a special focus on the pharmacological effects and possible mechanisms of GLP-1 mimetics in treating NAFLD/NASH, including dual and triple agonists at GLP-1R, glucose-dependent insulinotropic polypeptide receptor or glucagon receptor

The human gut bacteria Christensenellaceae are widespread, heritable, and associated with health

However, substantial fat regain typically accelerates over weeks and months because GLP-1 signalingwhich suppresses hunger and regulates energy expenditurestops completely

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These results strongly support the need for larger, randomized clinical trials to confirm these effects and explore the potential of repurposing GLP-1 RAs as a novel treatment for reducing harmful alcohol consumption

Biomarkers of inflammation decreased with sema treatment