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fenfluramine was subsequently withdrawn from the US market in fall of 1997
Its defining pharmacological feature is its simultaneous multi-pathway mechanism engaging the nitric oxide (NO) system, VEGFR2-mediated angiogenic signalling, and FAK-paxillin cytoskeletal signalling concurrently producing a broad cytoprotective and repair-promoting profile spanning gastrointestinal mucosal protection, tendon and ligament repair, muscle healing, bone regeneration, angiogenesis, neuroprotection, and wound healing
Viibryd (vilazodone) [prescribing information]
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