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If pancreatitis is confirmed, GLP-1 use must be stopped

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Preclinical studies suggest that GLP-1RAs have a beneficial effect on major NAFLD histological outcomes, i.e., hepatic steatosis and inflammation, through multiple intrahepatic mechanisms, including increased fatty acid -oxidation, activation of autophagy, suppression of inflammation, and oxidative stress

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But CNI inhibitor-induced excess secretion of ULVWF strings appears to overwhelm the capacity of ADAMTS-13 activity, thereby leading to platelet thrombi and TTP