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& Holness, M

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Nevertheless, the hope for therapeutic benefit from such agents, especially in the large fraction of patients refractory to established treatment approaches, drives continued interest (Pittenger, Coric et al., 2008

generated a double-knockout murine model by crossing the G6PD-deficient mice of Pretsch and Charles [62] with the atherosclerosis-prone apo E / mouse [12] and observed reduced aortic superoxide production, lower markers of inflammation, and less atheroma development

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