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The subdued reaction of Th17 cells to IL-2, combined with alterations in PI3K/Akt/mTORC1 activation cues, explain the hampered differentiation of these cells in the absence of GLS (111)

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Metabolic regulation of cholestatic liver injury by D-2-hydroxyglutarate with the modulation of hepatic microenvironment and the mammalian target of rapamycin signaling

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