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The arguments include the same dose range and same equipotent routes of application, regardless of the injury tested (Sikiric et al., 1993
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Preclinical evidence highlights stronger tendon structure and improved load-bearing capacity compared to untreated recovery conditions, concluding with a reference to BPC-157 for Tendon
Doxorubicin generates oxidative stress, damages mitochondria in heart cells, and triggers apoptosis (programmed cell death)
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