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This observation is consistent with earlier findings on DXN toxicity ( To ascertain the occurrence of cognitive impairment in rats administered with DXN, several behavioral assessments were conducted to evaluate hippocampal-dependent pathways involved in short-term memory encoding

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doi: 10.1080/713608372 30

The study lasted 9 weeks, during which clinical signs associated with biotin deficiency and biochemical parameters, such as serum and tissue biotin levels (liver, kidney, brain, testes), and biotin-dependent enzyme activity of 3-methylcrotonyl CoA carboxylase (MCC), propionyl-CoA carboxylase (PCC), pyruvate carboxylase (PC) and acetyl-CoA carboxylase (ACC), and blood glucose levels, were evaluated