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los mecanismos documentados corresponden a cada componente por separado, principalmente en modelos preclnicos (roedores e in vitro): BPC-157 : en estudios preclnicos se ha observado que modula la cicatrizacin a travs de la angiognesis, con up-regulacin temprana de genes como Vegfa , Nos3 y Egr1 , el depsito de colgeno, la actividad de fibroblastos y la va del xido ntrico, adems de una accin moduladora sobre clulas inflamatorias (Seiwerth et al., Front Pharmacol 2021

Research in cell culture and rodent models consistently demonstrates that MOTS-c accelerates this translocation independently of insulin, offering a distinct and additive mechanism to insulin sensitizers like Metformin [1]

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